Abstract
The idiopathic nephrotic syndrome (NS) in children is usually defined by the presence of heavy proteinuria, hypoalbuminemia, edema and moderate to severe hyperlipidemia (1). Hyperlipidemia Type 2B with increase in serum cholesterol and triglycerides is the most common and recognized metabolic complication in patients with NS. However, the precise mechanisms involved in its pathogenesis, the associated clinical complications of hyperlipidemia and the best approach for its management are not well established.