Abstract
As some of our research1 was marked by a large number of data and issues, some findings still have not been published; these are addressed in this chapter. The influence of cardiopulmonary bypass (CPB) itself on the inflammatory role played by some cell markers as well as the eventual impact of lung perfusion on this kind of inflammatory response were assessed using data obtained from hemograms, arterial blood gas analyses, and pulmonary leukosequestration. The whole hemogram was obtained from blood collected from the external jugular vein, and arterial blood for gas analysis was withdrawn from the internal carotid artery. Pulmonary leukosequestration was expressed as the ratio of the right atrial and left atrial white blood cell counts as postulated in a similar manner by Liu et al.2 Some authors have postulated that CPB is responsible for the significant rate of leukosequestration postoperatively, particularly in the lungs.3-5
