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A novel ASXL1-OGT axis plays roles in H3K4 methylation and tumor suppression in myeloid malignancies
Journal article   Peer reviewed

A novel ASXL1-OGT axis plays roles in H3K4 methylation and tumor suppression in myeloid malignancies

Daichi Inoue, Takeshi Fujino, Paul Sheridan, Yao-Zhong Zhang, Reina Nagase, Sayuri Horikawa, Zaomin Li, Hirotaka Matsui, Akinori Kanai, Makoto Saika, …
Leukemia, Vol.32(6), pp.1327-1337
2018-06
PMID: 29556021

Abstract

Animals Cell Differentiation Female HEK293 Cells Histones - metabolism HL-60 Cells Humans Leukemia, Myeloid, Acute - etiology Leukemia, Myeloid, Acute - prevention & control Methylation Mice Mice, Inbred C57BL Myelodysplastic Syndromes - etiology Myelodysplastic Syndromes - prevention & control N-Acetylglucosaminyltransferases - physiology Protein Stability Repressor Proteins - chemistry Repressor Proteins - physiology Tumor Suppressor Proteins - physiology

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Collaboration types
Domestic collaboration
International collaboration
Citation topics
1 Clinical & Life Sciences
1.103 Blood Disorders
1.103.155 Acute Myeloid Leukemia
Web Of Science research areas
Hematology
Oncology
ESI research areas
Clinical Medicine

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#3 Good Health and Well-Being

Source: InCites

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