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Breathing-Controlled Electrical Stimulation (BreEStim) Selectively Modulates Affective and Cognitive Components of Pain—An EEG Study
Journal article   Open access   Peer reviewed

Breathing-Controlled Electrical Stimulation (BreEStim) Selectively Modulates Affective and Cognitive Components of Pain—An EEG Study

Ahmad Z. Rao, Michael Houston, Hao Meng, Shengai Li, Yingchun Zhang and Sheng Li
Bioengineering (Basel), Vol.13(5), p.501
2026-04-25
PMID: 42194258

Abstract

breathing-controlled stimulation cortical processing event-related potentials pain modulation
Breathing-controlled electrical stimulation (BreEStim) is an innovative neuromodulation intervention that synchronizes deep voluntary breathing with peripheral electrical stimulation. Prior studies have shown its analgesic effects in healthy adults and spinal cord injury patients with neuropathic pain. The present study used EEG to examine BreEStim’s neural effects on sensory, affective, and cognitive components of pain. Fourteen healthy participants (7 M, 7 F) completed 30 min of BreEStim and conventional electrical stimulation (EStim) interventions in a randomized, crossover within-subject design. Electrical pain thresholds (EPT) and EEG were recorded pre- and post-intervention. Event-related potentials (ERPs) at pre-EPT-level stimuli before and immediately after each intervention were analyzed for early sensory (P30) and affective (P250) processing, while resting-state EEG assessed spectral power across delta, theta, alpha, and beta bands for cognitive processing. Both BreEStim and EStim increased EPT, indicating short-term habituation. There was no change in early ERP responses (P30) after each intervention, suggesting preserved sensory perception. BreEStim selectively reduced P250, reflective of the affective component of pain. BreEStim significantly increased delta and theta band power and reduced alpha band power on resting-state EEG analyses, whereas no significant changes after EStim were observed. Collectively, BreEStim preserves sensory encoding while selectively modulating affective and cognitive dimensions of pain, supporting its potential as a targeted, non-pharmacological neuromodulation strategy.
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https://doi.org/10.3390/bioengineering13050501View
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