Abstract
Although intracerebral hemorrhage is a less frequent cause of stroke than cerebral infarction, it is more often fatal. It is usually attributed to hypertensive small-vessel disease, and the most common sites of hemorrhage are the basal ganglia, cerebellum, and pons. Treatment is largely restricted to the control of hypertension and rehabilitation. In some patients with intracerebral hemorrhage, however, the hemorrhage is lobar in location, such as in the frontal, parietal, temporal, or occipital cortex, and such patients often do not have hypertension.
1
This category of hemorrhage, referred to as lobar intracerebral hemorrhage, may represent a distinct pathogenetic subgroup.
Cerebral amyloid . . .
