Nicotine attenuates iNOS expression and contributes to neuroprotection in a compressive model of spinal cord injury

Journal article

Peer reviewed

Journal of neuroscience research, Vol.87(4), pp.937-947

2009-03

PMID: 18855939

Abstract

inflammatory responses

neuronal nicotinic receptors

nicotine

nitric oxide synthase

oxidative stress

spinal cord injury

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Citation topics: 1 Clinical & Life Sciences; 1.82 Gait & Posture; 1.82.875 Spinal Cord Injury
Web Of Science research areas: Neurosciences
ESI research areas: Neuroscience & Behavior

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Source: InCites

Title: Nicotine attenuates iNOS expression and contributes to neuroprotection in a compressive model of spinal cord injury
Creators: Mee-Young Lee - Molecular Neuroscience and Vascular Biology Laboratory, Department of Neurosurgery, University of Kentucky, Lexington, Kentucky
Lei Chen - Molecular Neuroscience and Vascular Biology Laboratory, Department of Neurosurgery, University of Kentucky, Lexington, Kentucky
Michal Toborek - Molecular Neuroscience and Vascular Biology Laboratory, Department of Neurosurgery, University of Kentucky, Lexington, Kentucky
Publication Details: Journal of neuroscience research, Vol.87(4), pp.937-947
Publisher: Wiley Subscription Services, Inc., A Wiley Company
Number of pages: 11
Academic Unit: Miller School of Medicine; Biochemistry & Molecular Biology Research; UMMG Department of Surgery
Language: English
Resource Type: Journal article
PMID: 18855939
Record Identifier: 991031615554402976