Old mice retain bone marrow B1 progenitors, but lose B2 precursors, and exhibit altered immature B cell phenotype and light chain usage

Journal article

Open access

Peer reviewed

Mechanisms of ageing and development, Vol.130(6), pp.401-408

2009

PMID: 19428460

Abstract

Fundamental and applied biological sciences. Psychology

Development. Metamorphosis. Moult. Ageing

Biological and medical sciences

Vertebrates: anatomy and physiology, studies on body, several organs or systems

url

https://doi.org/10.1016/j.mad.2009.04.001View

Published (Version of record) Open

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Citation topics: 1 Clinical & Life Sciences; 1.6 Immunology; 1.6.452 Somatic Hypermutation
Web Of Science research areas: Cell Biology; Geriatrics & Gerontology
ESI research areas: Molecular Biology & Genetics

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Title: Old mice retain bone marrow B1 progenitors, but lose B2 precursors, and exhibit altered immature B cell phenotype and light chain usage
Creators: Sarah ALTER-WOLF - Department of Microbiology and Immunology, University of Miami Miller School of Medicine, Miami, FL 33101, United States
Bonnie B BLOMBERG - Department of Microbiology and Immunology, University of Miami Miller School of Medicine, Miami, FL 33101, United States
Richard L RILEY - Department of Microbiology and Immunology, University of Miami Miller School of Medicine, Miami, FL 33101, United States
Publication Details: Mechanisms of ageing and development, Vol.130(6), pp.401-408
Publisher: Elsevier; Shannon
Academic Unit: Miller School of Medicine; Microbiology and Immunology Research
Language: English
Resource Type: Journal article
PMID: 19428460
Record Identifier: 991031560262202976