Abstract
Alzheimer's disease is the most common cause of dementia late in life, affecting approximately 8 percent of people who are 65 years of age or older.
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Clinically diagnosed Alzheimer's disease is preceded by gradual, progressive memory loss. Neuritic plaques
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and neurofibrillary tangles,
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the neuropathological hallmarks of Alzheimer's disease, have also been found in adults without dementia, suggesting that the neuronal deficits leading to Alzheimer's disease begin years before any clinical changes occur. New and potential treatments for dementia focus on slowing the progression of the disease rather than regenerating neural cells, making it important to identify at an early stage . . .
