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Possible therapy for ALS based on the cyanobacteria/BMAA hypothesis
Journal article

Possible therapy for ALS based on the cyanobacteria/BMAA hypothesis

Walter G Bradley
Amyotrophic lateral sclerosis, Vol.10 Suppl 2(S2), pp.118-123
2009
DOI: https://doi.org/10.3109/17482960903285951
PMID: 19929743

Abstract

Amino Acids, Diamino - toxicity Amino Acids, Dicarboxylic - metabolism Amyotrophic Lateral Sclerosis - therapy Amino Acyl-tRNA Synthetases - drug effects Cyanobacteria - chemistry Humans Hair - chemistry Amyotrophic Lateral Sclerosis - diagnosis Neurotoxins - toxicity Amino Acids, Dicarboxylic - toxicity Protein Transport - drug effects Amino Acids, Diamino - analysis Neurotoxins - analysis Enzyme Inhibitors - therapeutic use Amino Acids - therapeutic use Cyanobacteria - physiology Amino Acids, Dicarboxylic - analysis Neurotoxins - metabolism Amyotrophic Lateral Sclerosis - chemically induced Amino Acids, Diamino - metabolism Amino Acyl-tRNA Synthetases - antagonists & inhibitors
Although the cyanobacteria/BMAA hypothesis of the cause of ALS and other age-related neurodegenerative diseases remains to be proven, it is not too early to ask whether treatment would be possible if the hypothesis were correct. This paper reviews the possible ways that chronic BMAA neurotoxicity could be prevented or treated.

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Citation topics
1 Clinical & Life Sciences
1.52 Neurodegenerative Diseases
1.52.765 Amyotrophic Lateral Sclerosis
Web Of Science research areas
Clinical Neurology
ESI research areas
Neuroscience & Behavior

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